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Subject: Re: is it RA?
Nearly all studies point to RA (and other autoimmune diseases) being triggered and sustained by cross reaction from gut bacteria. NSAID can relieve pain but also increase gut permeability causing increases in microbes already challenging our failing immune system. http://www.sciencedirect.com/science/article/pii/S0022175914000878 "Recently, Iwatsuki et al. (Iwatsuki et al., 2011) analyzed intestinal bacteria flora in elderly volunteers treated by oral administration of a natural milk antibody product, which contains biologically active antibodies against a wide range of pathogenic bacteria and their toxins (Kijima et al., 2009), and found significant reduction in the fecal population of pathogenic enteromicrobes such as E. coli, Clostridium difficile and perfringens. This approach was further applied for the treatment of patients with RA, resulting in modulated disease activities in 50% of these RA patients (Katayama et al., 2011). Together, these data support the idea that commensals (gut bacteria) and their toxins may contribute to the pathogenesis of autoimmune diseases." The treatments should keep this in mind - as antigen presentation (tagging proteins for tolerance) is informed by other bacteria in the gut. > Fix and maintain gut barrier with diet changes and limit NSAID use > Improve levels of gut bacteria that may be missing - that assist with accurate / tolerant / non-cross reactive antigen presentation > Reduce levels of potential bacterial (or sometime dietary) antigens (Proteus, E-Coli) that create cross reacting antibodies to people with specific HLA genes
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